This forces astrocytes to rely more on glycolysis for energy, contributing to metabolic reprogramming (Liddelow et al., 2017
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This special binding could be explained by the colocalization of mGluR1 and A and the presence of A and GluN2B immunoreactivities in neurites [35, 36] In AD, AO prevents physiological activation of the cellular prion protein-mGluR5 (PrPC-mGluR5) complex by glutamate
16 Gout prevalence increased with age and was more common among males
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This step establishes the baseline chemical environment required to keep GSH reduced