doi:10.1007/s10815-018-1225-2 [21] Aledo JC
However, this extrapolation from one organ to another is questionable as the exposure levels, the phase I metabolic capacity to generate NAPQI, the phase II metabolic capacity to eliminate APAP, and potential adaptive mechanisms in response to an APAP-induced cellular stress are not well described in the CNS system
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Miller M, Mead LA, Kwiterovich PO Jr, Pearson TA
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The metabolic condition is known to raise the risk of developing the memory-robbing disease by up to 77 percent
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