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Park, and W.K

Some patients respond very well, while others experience side effects, cost barriers, medication shortages, insurance denials, or weight regain after stopping therapy

While GLP-1RAs can directly stimulate mTORC1 in some contexts, the net effect of the impaired mTOR signaling during sustained caloric deficit may favor catabolism [34]

GLP-1 receptor agonists work by mimicking the natural hormone GLP-1, which is produced in the intestines