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Dual depletion of NHE1 and SWELL1 markedly suppresses breast cancer cell migration, extravasation, and metastasis, underscoring the pivotal role of this migration mechanism in breast cancer progression [163]

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The trial established the IGF-1 axis as a clinical therapeutic target and supports the broader pharmacological rationale for IGF-1 receptor agonism, of which IGF-1 LR3 is one analog with extended pharmacokinetic properties

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Protein biosynthesis, a target of sorafenib, interferes with the unfolded protein response (UPR) and ferroptosis in hepatocellular carcinoma cells

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