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Concomitant administration of CYP3A4 inhibitors (e.g., azole antifungals, macrolide antibiotics) or inducers (e.g., rifampin, St

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It can also activate extracellular signal-regulated kinase (ERK) and c-Jun N-terminal kinase (JNK), upregulating the proliferation-related gene expression in immune cells

[20] This common chemistry allows cells to use a small set of metabolic intermediates to carry chemical groups between different reactions

Instead of aiming at inhibition or modulation of the traditional targets of antibiotics, attempts are being made to identify pathways necessary for the microbes virulence (i.e., ability to cause a disease or damage) and the pathogenesis (how the infection develops to a disease) [71]